WebbTobias BOPP Professor (Full) for Molecular Immunology Univ-Prof. Dr. rer. nat. Johannes Gutenberg-Universität Mainz, Mainz JGU Institute for Immunology … Webb8 nov. 2024 · Tobias Bopp has received consulting fee and honoraria for lectures from Biogen, Celgene, Merck, Novartis, Pathios Therapeutics, Roche, Sanofi Genzyme, and Teva. Availability of data and materials Data will be provided upon reasonable request.
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WebbTobias Bopp Immunology, JGU Mainz „For me, EMTHERA provides the unique opportunity to work together with excellent scientists in an interdisciplinary fashion on tomorrow's therapies for diseases that are currently incurable.“ WebbTobias Bopp Professor bei University of Mainz Frankfurt Rhine-Main Metropolitan Area Insitute for Immunology, Universitätsmedizin Mainz, +1 more Tobias Bopp Multi-discipline engineer, Strategic...
Webb10 sep. 2024 · DOI: 10.1016/j.isci.2024.103092 Corpus ID: 238420347; The gut microbiota instructs the hepatic endothelial cell transcriptome @article{Formes2024TheGM, title={The gut microbiota instructs the hepatic endothelial cell transcriptome}, author={Henning Formes and Joana P. Bernardes and Amrit Mann and Franziska Bayer and Giulia … Webb5 nov. 2024 · TAMs in B16 tumors and human skin cutaneous melanoma are found to have high expression of ICER. Consistent with that, bone marrow–derived macrophages (BMDMs) robustly respond to organic acids (i ...
WebbUniv.-Prof. Dr. rer. nat. Tobias Bopp General Information E-Mail [email protected] Current position Professor for Molecular Immunology, W3 Academic Education 2003 … Webb26 jan. 2024 · Tobias Bopp 2Institute for Immunology, University Medical Center, Johannes Gutenberg University Mainz, Mainz, Germany Find articles by Tobias Bopp Christopher R. Parish 1The John Curtin School of Medical Research, The Australian National University, Canberra, ACT Australia Find articles by Christopher R. Parish Anne Brüstle
Webb22 dec. 2024 · Primary atopic disorders are a group of inborn errors of immunity that skew the immune system toward severe allergic disease. Defining the biology underlying these extreme monogenic phenotypes reveals shared mechanisms underlying common polygenic allergic disease and identifies potential drug targets.
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